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3.2 Mitochondrial metabolites as immune signaling molecules In HIV infection, mitochondrial dysfunction alters the levels of key metabolic intermediates, converting them from homeostatic signaling molecules into drivers of pathology

The table below represents a clinical summary of the two therapies across key categories: Primary mechanism: GHK-Cu acts as a tissue repair signal

doi: 10.2174/187221507779814461 231 ShenPDengXChenZBaXQinKHuangYet al

Therefore, the glucuronic acid derivative molecule is more readily excreted in both urine or bile